MiR-22 in tumorigenesis

SongS.J.; PandolfiP.P.

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MiR-22 in tumorigenesis

机译：MiR-22在肿瘤发生中的作用

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In recent years, it has become evident that lymphocyte are particularly adapted to form short-lived intercellular connections with the cells they scan. Such connections allow the transfer of membrane-anchored proteins, plasma membrane (PM) fragments, and even microRNAs.1"5 This "unorthodox" intercellular information transfer occurs through various incompletely defined mechanisms. For example, during the formation of an immunologi-cal synapse, lymphocytes snatcji PM fragments (also termed trogocytosis) and cell surface proteins from antigen-presenting cells (APCs).

机译：近年来，很明显，淋巴细胞特别适合与它们扫描的细胞形成短暂的细胞间连接。这种连接允许膜锚定蛋白，质膜（PM）片段甚至microRNA的转移。1“ 5这种“非正统”的细胞间信息转移通过各种不完全限定的机制发生，例如，在免疫学过程中突触，淋巴细胞snatcji PM片段（也称为trogocytosis）和来自抗原呈递细胞（APC）的细胞表面蛋白。

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《Cell cycle》 |2014年第1期|共2页
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SongS.J.; PandolfiP.P.;
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正文语种 eng
中图分类细胞生物学;
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4. Tumorigenesis and metastasis scheme from photodynamic therapy perspective [C] . A.S. Skobeltcin, A.V. Ryabova, Yu. S. Maklygina, International Conference Laser Optics . 2020

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5. Role of Interleukin-1 Receptor-Associated Kinases in Chronic Inflammation and Prostate Tumorigenesis [D] . Oseni, Saheed Oluwasina. 2021

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6. miR-22 suppresses tumorigenesis and improves radiosensitivity of breast cancer cells by targeting Sirt1 [O] . Xia Zhang, Yuehua Li, Dan Wang, 2017

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7. Suppression of miR-22, a tumor suppressor in cervical cancer, by human papillomavirus 16 E6 via a p53/miR-22/HDAC6 pathway [O] . Weerayut Wongjampa, Tipaya Ekalaksananan, Peechanika Chopjitt, 2018

机译：通过P53 / miR-22 / HDAC6途径抑制miR-22，宫颈癌中的肿瘤抑制剂，通过人乳头瘤病毒16e6

MiR-22 in tumorigenesis

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