METHOD FOR STORING ACORNS OF COMMON OAK

• 1. 用于操纵带有胎圈－三角胶条和间隔件的多个堆叠的胎圈－三角胶条储存系统和方法 [P] . 中国专利： CN112009002A . 2020-12-01
• 2. 一种普通外科护理用药品储存装置 [P] . 中国专利： CN215045015U . 2021-12-07
• 3. LATTICE FOR TREATING ACORNS OF COMMON OAK (QUERCUS ROBUR L.) [P] . UA73042U . 2012-09-10

  机译： 用于处理普通橡树皮（QUERCUS ROBUR L.）的格子

• 4. METHOD OF SOWING ACORNS INTO CONTAINERS WHILE GROWING OAK SEEDLINGS WITH CLOSED ROOT SYSTEM [P] . 俄罗斯专利： RU2621993C1 . 2017-06-08

  机译： 封闭根系种植橡树苗时将玉米播种到容器中的方法。

• 5. METHOD FOR TREATING ACORNS OF THE PEDUNCULATE OAK (QUERCUS ROBUR L.) WITH BEESWAX [P] . UA79995U . 2013-05-13

  机译： 用蜂蜡处理带蒂橡木（QUERCUS ROBUR L.）角的方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Biorational control methods for protection of stored grain legumes against bruchid beetles [J] . Takashi Yamane . 农业科学（英文） . 2013,第12期
• 3. 普通胰岛素注射液启封后储存方式的研究进展 [C] . 马颖颖 ,李沛 ,姜泓 . 2017中国护理管理大会 . 2017
• 4. 深冻锅与普通烹饪蔬菜在不同储存条件下硝酸盐、亚硝酸盐及维生素C含量的比较 [A] . 陈若文 . 2016