BRONCHIOPULMONARY OCCLUSION DEVICES AND LUNG VOLUME REDUCTION METHODS

• 1. 一种直接测量动物肺体积和间接测量肺密度的装置 [P] . 中国专利： CN213940732U . 2021-08-13
• 2. 可拆卸型肺减容支气管栓子的设计方法及装置 [P] . 中国专利： CN1726873A . 2006-02-01
• 3. Bronchiopulmonary occlusion devices and lung volume reduction methods [P] . AU2001260840B2 . 2005-08-11

  机译： 支气管肺阻塞装置和减少肺体积的方法

• 4. BRONCHIOPULMONARY OCCLUSION DEVICES AND LUNG VOLUME REDUCTION METHODS [P] . 欧洲知识产权局专利： EP1284663A1 . 2003-02-26

  机译： 支气管肺闭塞装置和肺体积减少方法

• 5. BRONCHIOPULMONARY OCCLUSION DEVICES AND LUNG VOLUME REDUCTION METHODS [P] . 世界知识产权组织专利： WO0187170A1 . 2001-11-22

  机译： 支气管肺闭塞装置和肺体积减少方法

• 1. NUMERICAL METHOD OF MIXED FINITE VOLUME-MODIFIED UPWIND FRACTIONAL STEP DIFFERENCE FOR THREE-DIMENSIONAL SEMICONDUCTOR DEVICE TRANSIENT BEHAVIOR PROBLEMS [J] . Yirang YUAN, Qing YANG, Changfeng LI, 数学物理学报（英文版） . 2017,第001期
• 2. Bronchial Occlusion with Endobronchial Watanabe Spigot (EWS) on Pulmonary Diseases and Bronchoscopic Lung Volume Reduction (BLVR) [C] . Watanabe Y., Fujii H., Morichika D., World Congress for Bronchology . 2011
• 3. Effect of acute lung volume reduction surgery on lung mechanics and maximal flow in a canine model of upper lobe emphysema. [D] . Buduhan, Gordon. 2005
• 4. Lung volume reduction surgery or bronchoscopic lung volume reduction: is there an algorithm for allocation? [O] . Matthew Gordon, Sean Duffy, Gerard J. Criner 2018
• 5. Poster Session 3The imaging examination and quality assessmentP626Value of mitral and tricuspid annular displacement to assess the interventricular systolic relationship in severe aortic valve stenosis : a Pilot studyP627Follow-up echocardiography in asymptomatic valve disease: assessing the potential economic impact of the European and American guidelines in a dedicated valve clinic, compared to standard care.P628The tricuspid valve: identification of optimal view for assessing for prolapseP629Right atrial volume by two-dimensional echocardiography in healthy subjectsP630Disturbance of inter and intra atrial conduction assessed by tissue doppler imaging in patients with medicaly controlled hypertension and prehypertension.P631Liver stiffness by shear wave elastography, new noninvasive and quantitative tool for acute variation estimation of central venous pressure in real-time?P632Weak atrial kick contribution is associated with a risk for heart failure decompensationP633Usefulness of wave intensity analysis in predicting the response to cardiac resynchronization therapyP634Early subclinical left ventricular systolic and diastolic dysfunction in gestational hypertension and preeclampsiaP635Clinical comparison of three different echocardiographic methods for left ventricular ejection fraction and LV end diastolic volume measurementP636Assessment of right ventricular-arterial coupling parameters by 3D echocardiography in patients with pulmonary hypertension receiving specific vasodilator therapyP637Prediction of right ventricular failure after left ventricular assist device implant: assessing usefulness of standard and strain echocardiographyP638Kinematic analysis of diastolic function using the novel freely available software Echo E-waves - feasibility and reproducibilityP639Evaluation of coronary flow velocity by Doppler echocardiography in the treatment of hypertension with the ARB: correlation to the histological cardiac fibrosisP640The clinical significance of limited apical ischaemia and the prognostic value of stress echocardiography - A contemporary study from a high volume centerP641Effects of intermediate stenosis of left anterior descending coronary artery on survival in patients with chronic total occlusion of right coronary arteryP642Left ventricular remodeling after a first myocardial infarction in patients with preserved ejection fraction at dischargeP643Left atrial size and acute coronary syndromes. Let is make simple.P644Influence of STEMI reperfusion strategy on systolic and diastolic functionP645Aortic valve resistance risk-stratifies low-gradient severe aortic stenosisP646Does permanent pacemaker implantation complicate the prognosis of patients after transcatheter aortic valve implantation?P647Influence of metabolic syndrome and diabetes on progression of calcific aortic valve stenosis - The COFRASA - GENERAC StudyP648Low referral for aortic valve replacement accounts for worse long-term outcome in low versus high gradient severe aortic stenosis with preserved ejection fractionP649The impact of right ventricular function from aortic valve replacement: A randomised study comparing minimally invasive aortic valve surgery and conventional open heart surgery [O] . T. Ota, DNS Senaratne, NK. Preston, 2016

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Effect of lung volume reduction surgery on early function of lung and pulmonary hemodynamics in COPD [J] . 侯生才 . 外科研究与新技术 . 2003,第2期
• 3. Percutaneous devices for left atrial appendage occlusion: A contemporary review [J] . Homam Moussa Pacha ,Yasser Al-khadra ,Mohamad Soud . 世界心脏病学杂志：英文版（电子版） . 2019,第2期
• 4. Randomized study comparing incidence of radial artery occlusion post-percutaneous coronary intervention between two conventional compression devices using a novel airinflation technique [J] . Victor Voon ,Muhammad Ayyaz Ul Haq ,Ciara Cahill . 世界心脏病学杂志：英文版（电子版） . 2017,第11期
• 5. 不同肺体积确定方法对肺剂量体积参数的影响 [C] . 徐利明 ,谭文勇 ,李承军 . 第二届泛珠江三角区放射肿瘤学术大会暨四川省第四届放射肿瘤专委会第三届年会 . 2007
• 6. 肺结节二维及三维体积测量方法在肺结节体积及体积变化百分比测量中观察者内的重复性比较研究 [A] . 郭小皖 . 2014