FREEZING AND DEFROSTING STORED WATER FOR USE IN RITUAL BATH ACCORDING TO HALACHA

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• 2. 用于冷冻水列间空调的控制方法及装置、冷冻水列间空调 [P] . 中国专利： CN113959055A . 2022-01-21
• 3. FREEZING AND DEFROSTING STORED WATER FOR USE IN RITUAL BATH ACCORDING TO HALACHA [P] . IL148499D0 . 2002-09-12

  机译： 冷冻和除霜储存的水，用于根据哈拉查节举行的仪式浴

• 4. WATER PURIFIER AND A METHOD FOR CLEANING THE WATER STORING BATH OF THE WATER PURIFIER CAPABLE OF INCLUDING A CLEANING FUNCTION WITH RESPECT TO THE WATER STORING BATH OF THE WATER PURIFIER [P] . 韩国专利： KR20110094171A . 2011-08-22

  机译： 水净化器和一种清洁水净化器的方法，能够包括关于水净化器的储水槽的清洁功能

• 5. WATER FILTER FOR RITUAL BATH [P] . IL167627A . 2006-02-05

  机译： 仪式浴滤水器

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Levels of Trihalomethanes in Stored Water from High and Fundamental Schools:Comparison between Two Temporal Data Sets [J] . Ricardo Andreola ,Anny Rosi Mannigel ,Graciene de Souza Bido . 水资源与保护（英文） . 2018,第6期
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• 5. Experimental Model to Treat BOD5, COD in Wastewater by Eichhornia Crassipes Raft, and Propose a Plan to Restore Water Source of To Lich River, Hanoi, Vietnam [J] . Nguyen Thuc Hien1 ,Nguyen Minh Khuyen2 . 环境科学与工程：B . 2016,第004期
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