METHOD FOR PRODUCING UPPER DENTURE IN CASES OF CONGENITAL DEFECTS OF MAXILLARY ALVEOLAR CREST

• 1. 上颌牙槽突骨牵引器 [P] . 中国专利： CN206576943U . 2017.10.24
• 2. 一种隐形垂直向的上颌牙矫正装置及其使用方法 [P] . 中国专利： CN112773525A . 2021-05-11
• 3. METHOD OF PROSTHETICS IN PATIENTS WITH EXPRESSED CONGENITAL DEVELOPMENTAL ABNORMALITIES AND ACQUIRED DEFORMATIONS OF ALVEOLAR CRESTS OF UPPER AND LOWER JAW BONES [P] . 俄罗斯专利： RU2485911C1 . 2013-06-27

  机译： 先天性发育异常且上下颌骨牙槽C获得性畸形的假体修复方法

• 4. METHOD FOR REMOVING MAXILLARY ALVEOLAR PROCESS DEFECT IN PERFORMING PLASTIC REPAIR OF CLEFT LIP AND CONCURRENT NOSE SYMMETRY RESTORATION [P] . 俄罗斯专利： RU2134076C1 . 1999-08-10

  机译： 进行唇唇缘整形修复和同时保持鼻尖对称性的上颌最大牙周畸形的方法

• 5. METHOD FOR DISTAL ALVEOLAR PROCESS OF UPPER JAW IN CHRONIC POLYPOUS MAXILLARY SINUSITIS [P] . 俄罗斯专利： RU2714169C1 . 2020-02-12

  机译： 慢性多发性上颌窦窦上颌远端牙槽突的治疗方法

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• 1. Flapless Piezotome-Enhanced Vertical Alveolar Crest-Split and Horizontal Distraction of Alveolar Crests (FPeCSWT) of Less than 2 mm Width: Results of a Prospective Comparative 3-Year Clinical Multicenter-Study with 239 Patients, 261 Crest-Split Sites and 488 Inserted Dental Implants [J] . Angelo Troedhan ,Andreas Kurrek ,Marcel Wainwright . 口腔学期刊（英文） . 2015,第7期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 上颌骨重建方法学的选择——1067例病例分析报告 [C] . Liqun XU ,徐立群 ,Chenping ZHANG . 2013国际暨全国第十二届头颈肿瘤学术大会 . 2013
• 4. 种植体支抗整体后退上颌牙弓二次矫治支抗丧失病例的临床研究 [A] . Nakhonkham Bannavong 泰娴 . 2018