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• 1. 一种基于特征正交分解的非平稳随机过程快速模拟方法 [P] . 中国专利： CN107247687B . 2021.06.08
• 2. 一种基于平稳随机过程的信道相关阴影衰落构建的方法 [P] . 中国专利： CN103561412B . 2017.01.11
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  机译： 平稳随机过程的设备л参数光谱分析

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  机译： 随机极化周期信号的极化谱分析装置

• 5. HIGH SPEED STATIONARY FIELD ANALYSIS METHOD, HIGH SPEED STATIONARY FIELD ANALYSIS DEVICE, HIGH SPEED STATIONARY FIELD ANALYSIS PROGRAM AND COMPUTER-READABLE RECORDING MEDIUM WITH THE SAME PROGRAM RECORDED [P] . 日本专利： JP2012069024A . 2012-04-05

  机译： 高速静止场分析方法，高速静止场分析装置，高速静止场分析程序和具有相同记录程序的计算机可读记录介质

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Gradient Density Estimation in Arbitrary Finite Dimensions Using the Method of Stationary Phase [J] . Karthik S. Gurumoorthy ,Anand Rangarajan ,John Corring . 理论数学进展（英文） . 2019,第12期
• 3. 基于随机波和POD的非平稳随机过程的快速模拟 [C] . 赵宁 ,彭留留 ,黄国庆 . 第十八届全国结构风工程学术会议暨第四届全国风工程研究生论坛 . 2017
• 4. 一个连续时间非平稳随机过程的核密度估计量的大样本性质 [A] . 李长国 . 2001