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首页> 外文会议>Basic and Clinical Ocular Motor and Vestibular Research: A Tribute to R. John Leigh >Antisaccade generation is impaired after parietal lobe lesions
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Antisaccade generation is impaired after parietal lobe lesions

机译：榫栗叶病变后反剖解产生损害

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Antisaccades are directed away from visual targets. Impaired antisaccade generation has been attributed to frontal lobe damage. We studied antisaccades in patients with unilateral focal parietal lobe lesions. Normal subjects (N = 10) instructed to make 10° antisaccades opposite to a 100-ms target flash 10° to the right or left of center made antisaccades in 86.1% of trials. Patients (N = 13) made antisaccades contraversive to their lesions in 55.4% of trials and 50.5% of ipsiversive trials. In other trials, reflexive saccades occurred toward the target flash. Nine patients with imaged lesions overlapping in parietal lobe white matter showed subnormal antisaccade generation. Antisaccades provide a means of measuring voluntary saccade function of the parietal lobes independent of visual guidance. Impaired suppression of reflexive saccades and generation of antisaccades is attributed to disconnection of parietal lobe from frontal lobe ocular motor areas.
机译：反脱离行为偏离视觉目标。反剖解生成受损归因于额叶损伤。我们研究了单侧局灶性凸角病变患者的反肿瘤。正常受试者（n = 10）指示使10°反剖面与100 ms目标闪光10°的右侧或左侧的左侧或左侧的试验中的86.1％以86.1％的试验。患者（n = 13）对55.4％的试验和50.5％的IPsiversive试验中的病变进行了反腐病。在其他试验中，对目标闪光发生反射扫描。九个患者凸角白质重叠的成像病变显示了亚型反易分析。反脱离行为提供了一种衡量顶部裂片的自愿扫视功能的方法，与视觉指导无关。抑制反射扫描的抑制和反裂解的产生归因于断开前壁叶片与额叶眼镜电机区域的连接。

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