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Direct Impairment of Vascular Function by Diesel Exhaust Particulate through Reduced Bioavailability of Endothelium-Derived Nitric Oxide Induced by Superoxide Free Radicals

机译：柴油机排气微粒直接降低血管功能原因是超氧化物自由基诱导的内皮衍生的一氧化氮的生物利用度降低

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Background Diesel exhaust particulate (DEP) is a key arbiter of the adverse cardiovascular effects of air pollution.

机译：背景技术柴油机排气微粒（DEP）是空气污染对心血管的不利影响的关键仲裁者。

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期刊名称 Environmental Health Perspectives
作者
Mark R. Miller; Stephen J. Borthwick; Catherine A. Shaw; Steven G. McLean; Daniel McClure; Nicholas L. Mills; Rodger Duffin; Ken Donaldson; Ian L. Megson; Patrick W.F. Hadoke; David E. Newby;
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年(卷),期 2009(117),4
年度 2009
页码 611–616
总页数 6
原文格式 PDF
正文语种
中图分类公共卫生工程;
关键词
blood vessel diesel nitric oxide particulate pollution superoxide;

机译：血管;柴油;一氧化氮;微粒;污染;超氧化物;

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专利

1. Direct impairment of vascular function by diesel exhaust particulate through reduced bioavailability of endothelium-derived nitric oxide induced by superoxide free radicals. [J] . Miller MR, Borthwick SJ, Shaw CA, Environmental health perspectives. . 2009,第4期

机译：柴油机排气微粒直接降低血管功能，原因是超氧化物自由基诱导的内皮衍生的一氧化氮的生物利用度降低。
2. Naphthazarin and methylnaphthazarin cause vascular dysfunction by impairment of endothelium-derived nitric oxide and increased superoxide anion generation. [J] . Kang JJ, Lee PJ, Chen YJ, Toxicology in vitro: an international journal published in association with BIBRA . 2006,第1期

机译：萘达沙林和甲基萘达沙林通过破坏内皮衍生的一氧化氮和增加超氧阴离子的产生而引起血管功能障碍。
3. Dairy milk proteins attenuate hyperglycemia-induced impairments in vascular endothelial function in adults with prediabetes by limiting increases in glycemia and oxidative stress that reduce nitric oxide bioavailability [J] . McDonald Joshua D., Mah Eunice, Chitchumroonchokchai Chureeporn, The Journal of Nutritional Biochemistry . 2019,第期

机译：乳制品蛋白质通过限制糖血症和氧化应激的增加，减少血管内皮功能的高血糖内皮功能损伤，降低一氧化氮生物利用度
4. Vascular endothelial dysfunction with aging: nitric oxide bioavailability and tumor necrosis factor-alpha levels [C] . Eksakulkla S, Suksom D, Patumraj S, Asian Congress for Microcirculation . 2011

机译：血管内皮功能障碍与老化：一氧化氮生物利用度和肿瘤坏死因子 - α水平
5. INVOLVEMENT OF LIPID PEROXIDATION IN THE DEVELOPMENT OF BACTERIA-INDUCED HYPERSENSITIVE REACTION (LIPOXYGENASE, SUPEROXIDE DISMUTASE, FREE RADICALS, PARAQUAT). [D] . KEPPLER, LAVERNE DALE. 1986

机译：脂质引起的过氧化物参与细菌引起的超敏反应（脂氧合酶，超氧化物歧化酶，自由基，百草枯）的发展。
6. Altered Nitric Oxide Bioavailability Contributes to Diesel Exhaust Inhalation‐Induced Cardiovascular Dysfunction in Man [O] . Jeremy P. Langrish, Jon Unosson, Jenny Bosson, -1

机译：一氧化氮的生物利用度改变导致柴油机排气吸入引起的人的心血管功能障碍
7. Direct Impairment of Vascular Function by Diesel Exhaust Particulate through Reduced Bioavailability of Endothelium-Derived Nitric Oxide Induced by Superoxide Free Radicals [O] . Miller, Mark R., Borthwick, Stephen J., Shaw, Catherine A., 2009

机译：超氧化物自由基诱导内皮源性一氧化氮降低生物利用度对柴油机排气微粒造成血管功能的直接损害

Direct Impairment of Vascular Function by Diesel Exhaust Particulate through Reduced Bioavailability of Endothelium-Derived Nitric Oxide Induced by Superoxide Free Radicals

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