Reduced gastric acid production in burn shock period and its significance in the prevention and treatment of acute gastric mucosal lesions

Zhu L; Yang ZC; Li A

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Reduced gastric acid production in burn shock period and its significance in the prevention and treatment of acute gastric mucosal lesions

机译：烧伤休克期胃酸生成减少及其对急性胃粘膜病变的预防和治疗意义

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AIM To investigate the changes of gastric acid production and its mechanism in shock period of severe burn in rats. METHODS A rat model with 30/100 TBSA full- Thickness burn injury was employed and the Gastric acid production, together with gastric Mucosal blood flow (GMBF) and energy charge (EC) were measured serially within 48h postburn. RESULTS The gastric acid production in the Acute shock period was markedly inhibited after Severe burn injury.

机译：目的探讨重度烧伤休克期大鼠胃酸产生的变化及其机制。方法采用30/100 TBSA全层烧伤的大鼠模型，在烧伤后48小时内连续测量胃酸的产生，胃粘膜血流量（GMBF）和能量（EC）。结果严重烧伤后急性休克期胃酸的产生受到明显抑制。

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《World Journal of Gastroenterology》 |2000年第1期|p.84-88|共5页
作者
Zhu L; Yang ZC; Li A;
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收录信息美国《科学引文索引》(SCI);美国《工程索引》(EI);美国《生物学医学文摘》(MEDLINE);美国《化学文摘》(CA);
原文格式 PDF
正文语种 eng
中图分类消化系及腹部疾病;
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1. A critical role of gastric mucosal ascorbic acid in the progression of acute gastric mucosal lesions induced by compound 48/80 in rats. [J] . Kamiya Y, Ohta Y, Imai Y, World Journal of Gastroenterology . 2005,第9期

机译：胃粘膜抗坏血酸在化合物48/80诱导的大鼠急性胃粘膜损伤进展中的关键作用。
2. A change in gastric mucosal ascorbic acid status with the formation, progression, and recovery of compound 48/80-induced acute gastric mucosal lesions in rats. [J] . Ohta Y, Kamiya Y, Imai Y, Journal of Nutritional Science and Vitaminology . 2004,第5期

机译：胃粘膜抗坏血酸状态的变化，与化合物48/80诱导的大鼠急性胃粘膜病变的形成，进展和恢复有关。
3. Intestinal trefoil factor produced in Escherichia coli promotes the healing of rat burn-induced acute gastric mucosal lesions. [J] . Sun Y, Wu W, Zhang Y, The Journal of trauma . 2008,第1期

机译：大肠杆菌中产生的肠三叶因子促进大鼠烧伤诱导的急性胃粘膜病变的愈合。
4. Prevention of Ethanol-inclucecl Gastric Mucosal Microcirculatory Disturbances by Mild Irritant Through the Actions of Calcitonin Gene-Related Peptide and Prostaglandin l2 in Rats [C] . Takeo Saeki, Takashi Ohno, Kazuhisa Kamata, Keio International Symposia for Life Sciences and Medicine . 2005

机译：通过钙蛋白基因相关肽和前列腺素L2的慢性刺激物通过轻度刺激物预防乙醇 - 增链型胃粘膜微循环干扰
5. The relationship between gastric mucosal acidosis and the development of sepsis and organ failure in the critically ill. [D] . Just, Jon David. 1994

机译：危重病患者胃粘膜酸中毒与脓毒症和器官衰竭的发展之间的关系。
6. Reduced gastric acid production in burn shock period and its significance in the prevention and treatment of acute gastric mucosal lesions [O] . Li Zhu, Zhong-Cheng Yang, Ao Li, 2000

机译：烧伤休克期胃酸生成减少及其对急性胃粘膜病变的预防和治疗意义
7. Reduced gastric acid production in burn shock period and its significance in the prevention and treatment of acute gastric mucosal lesions [O] . Li Zhu 2000

机译：降低燃烧休克期的胃酸产生及其在急性胃黏膜病变预防和治疗中的意义

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Reduced gastric acid production in burn shock period and its significance in the prevention and treatment of acute gastric mucosal lesions

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