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首页> 外文期刊>Biology Open >Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin

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Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin

机译：结核分枝杆菌Cpn60.2（GroEL2）通过与线粒体mortalin相互作用阻止巨噬细胞凋亡结核分枝杆菌Cpn60.2（GroEL2）通过与线粒体mortalin相互作用阻止巨噬细胞凋亡

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cannot no abstract right now, maybe has no abstract or it's too hard to get

机译：现在不能没有摘要，可能没有摘要，或者太难获取

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《Biology Open》 |2017年第4期|共页
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Zakaria Hmama; Sunil Joseph; Alex Yuen; Vijender Singh;
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1. Mycobacterium tuberculosis?Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin [J] . Sunil Joseph, ?Alex Yuen, ?Vijender Singh, Biology Open . 2017,第4期

机译：结核分枝杆菌Cpn60.2（GroEL2）通过与线粒体mortalin的相互作用阻断巨噬细胞凋亡
2. Interaction mechanism of Mycobacterium tuberculosis GroEL2 protein with macrophage Lectin-like, oxidized low-density lipoprotein receptor-1: An integrated computational and experimental study [J] . Vinod Vivek, Pushkaran Anju Choorakottayil, Kumar Anil, Biochimica et Biophysica Acta. General Subjects . 2021,第1期

机译：结核分枝杆菌结核菌蛋白与巨噬细胞凝集素样蛋白的相互作用机理，氧化低密度脂蛋白受体-1：综合计算与实验研究
3. The 19?kDa Mycobacterium tuberculosis Lipoprotein (LpqH) Induces Macrophage Apoptosis through Extrinsic and Intrinsic Pathways: A Role for the Mitochondrial Apoptosis-Inducing Factor [J] . Alejandro Sánchez, Patricia Espinosa, Teresa García, Clinical & developmental immunology. . 2012,第1期

机译：19？kDa结核分枝杆菌脂蛋白（LpqH）通过外在和内在途径诱导巨噬细胞凋亡：线粒体细胞凋亡诱导因子的作用。
4. PPE2 protein of Mycobacterium tuberculosis may inhibit nitric oxide in activated macrophages [C] . Khalid Hussain Bhat, Arghya Das, Aparna Srikantam, Federation of Immunological Societies of Asia-Oceania. . 2013

机译：结核分枝杆菌的PPE2蛋白可能抑制活化巨噬细胞中的一氧化氮
5. Host immune response in tuberculosis: An examination of the interaction of Mycobacterium tuberculosis with dendritic cells and macrophages. [D] . Jang, Sihyug. 2004

机译：结核病中的宿主免疫反应：检查结核分枝杆菌与树突状细胞和巨噬细胞的相互作用。
6. The 19 kDa Mycobacterium tuberculosis Lipoprotein (LpqH) Induces Macrophage Apoptosis through Extrinsic and Intrinsic Pathways: A Role for the Mitochondrial Apoptosis-Inducing Factor [O] . Alejandro Sánchez, Patricia Espinosa, Teresa García, 2012

机译：19 kDa结核分枝杆菌脂蛋白（LpqH）通过外在和内在途径诱导巨噬细胞凋亡：线粒体细胞凋亡诱导因子的作用。
7. Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin [O] . Sunil Joseph, Alex Yuen, Vijender Singh, 2017

机译：结核分枝杆菌Cpn60.2（GroEL2）通过与线粒体mortalin的相互作用阻断巨噬细胞凋亡

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