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首页> 外文期刊>Case Reports in Nephrology >Clinicopathological Implications of Proteinuria after Long-Term Isolated Hematuria due to Thin Basement Membrane Nephropathy and Focal Segmental Glomerulosclerosis
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Clinicopathological Implications of Proteinuria after Long-Term Isolated Hematuria due to Thin Basement Membrane Nephropathy and Focal Segmental Glomerulosclerosis

机译：由于薄的基底膜肾病和局灶性节段性肾小球粥样硬化，蛋白尿后蛋白尿后蛋白尿后的临床病理学意义

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A 45-year-old obese man presented with persistent hematuria for 21 years. At the age of 37, he developed hypertension and proteinuria which later increased up to 1.6?g/g creatinine. Kidney biopsy revealed thin basement membrane nephropathy (TBMN) and focal segmental glomerulosclerosis (FSGS), which explained his urinary abnormalities. Although a subgroup of TBMN can be complicated by FSGS, his FSGS was associated with obesity because of its histological features. Reduction of body weight and increasing a dose of angiotensin-receptor blocker could transiently reduce the amount of proteinuria. Clinicopathological implications of proteinuria after long-term hematuria by TBMN and FSGS were further discussed.
机译：一个45岁的肥胖男子患有持续的血尿21年。在37岁时，他开发了高血压和蛋白尿，后来增加了1.6？G / G肌酐。肾活检显示薄的基底膜肾病（TBMN）和局灶性节段性肾小球粥样硬化（FSG），其解释了他的泌尿情况。虽然FSGs的TBMN的亚组可以复杂，但由于其组织学特征，他的FSGs与肥胖有关。减少体重和增加剂量的血管紧张素受体阻滞剂可以瞬时减少蛋白尿的量。进一步讨论了TBMN和FSGs后长期血尿后蛋白尿临床病理学影响。

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