...
首页> 外文期刊>Proceedings of the National Academy of Sciences of the United States of America >Heightened expression of tumor necrosis factor α, interleukin 1α , and glial fibrillary acidic protein in experimental Creutzfeldt-Jakob disease in mice
【24h】

Heightened expression of tumor necrosis factor α, interleukin 1α , and glial fibrillary acidic protein in experimental Creutzfeldt-Jakob disease in mice

机译:实验性Creutzfeldt-Jakob病小鼠的肿瘤坏死因子α,白介素1α和胶质纤维酸性蛋白的表达升高

获取原文
获取原文并翻译 | 示例
           

摘要

The ultrastructural pathology of myelinated axons in mice infected experimentally with the Fujisaki strain of Creutzfeldt-Jakob disease (CJD) virus is characterized by myelin sheath vacuolation that closely resembles that induced in murine spinal cord organotypic cultures by tumor necrosis factor α (TNF-α), a cytokine produced by astrocytes and macrophages.
机译:实验性感染Creutzfeldt-Jakob病(FJ)的Fujisaki株的小鼠的髓鞘轴突的超微结构病理学特征是髓鞘鞘空泡化,与肿瘤坏死因子α(TNF-α)在鼠脊髓器官典型培养物中诱导的髓鞘空泡化非常相似。 ,是星形胶质细胞和巨噬细胞产生的细胞因子。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱:kefu@zhangqiaokeyan.com

京公网安备:11010802029741号 ICP备案号:京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有
  • 客服微信

  • 服务号