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首页> 外文期刊>Trends in Plant Science >UV-B exposure, ROS, and stress: inseparable companions or loosely linked associates?
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UV-B exposure, ROS, and stress: inseparable companions or loosely linked associates?

机译：UV-B暴露，ROS和压力：密不可分的伴侣或联系松散的伙伴？

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Ultraviolet-B (UV-B) radiation has long been perceived as a stressor. However, a conceptual U-turn has taken place, and UV-B damage is now considered rare. We question whether UV-stress and UV-B-induced reactive oxygen species (ROS) are still relevant concepts, and if ROS-mediated signaling contributes to UV-B acclimation. Measurements of antioxidants and of antioxidant genes show that both low and high UV-B doses alter ROS metabolism. Yet, there is no evidence that ROS control gene expression under low UV-B. Instead, expression of antioxidant genes is linked to the UV RESISTANCE LOCUS 8 pathway. We hypothesize that low UV-B doses cause 'eustress' (good stress) and that stimuli-specific signaling pathways pre-dispose plants to a state of low alert that includes activation of antioxidant defenses.
机译：长期以来，人们一直将紫外线B（UV-B）辐射视为压力源。但是，发生了概念性的掉头，现在认为UV-B损坏很少。我们质疑紫外线胁迫和紫外线B诱导的活性氧（ROS）是否仍然是相关概念，以及ROS介导的信号传导是否有助于紫外线B适应。对抗氧化剂和抗氧化剂基因的测量表明，低剂量和高剂量的UV-B都会改变ROS的代谢。然而，没有证据表明ROS在低UV-B下控制基因表达。相反，抗氧化剂基因的表达与UV RESISTANCE LOCUS 8途径相关。我们假设低剂量的UV-B会引起“胁迫”（良好胁迫），并且刺激特异性信号传导途径会使植物处于低警戒状态，包括激活抗氧化剂防御能力。

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