Mitochondrial mutations may decrease population viability

Gemmell NJ.; Allendorf FW.

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Mitochondrial mutations may decrease population viability

机译：线粒体突变可能会降低种群活力

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It is ironic that an organelle that is pivotal for the function of male gametes is inherited by sons only from their mother. A recent study of human fertility has provided the first definitive evidence that mitochondrial DNA mutations can reduce male fertility but with little or no effect on females, These results present the possibility that the viability of small populations might be reduced by increases in the frequency of mtDNA genotypes that lower the fitness of males. [References: 12]

机译：具有讽刺意味的是，对于雄配子功能至关重要的细胞器仅由儿子从母亲那里继承。一项有关人类生育力的最新研究提供了第一个明确的证据，即线粒体DNA突变可以降低男性的生育能力，但对女性的影响很小或没有影响。这些结果表明，通过增加mtDNA的频率可能降低小种群的生存能力降低男性健康水平的基因型。 [参考：12]

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《Trends in Ecology & Evolution》 |2001年第3期|共3页
作者
Gemmell NJ.; Allendorf FW.;
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正文语种 eng
中图分类生态学（生物生态学）;
关键词
Motility; Sperm;

机译：动力;精子;

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1. Mitochondrial mutations may decrease population viability [J] . Gemmell NJ., Allendorf FW. Trends in Ecology & Evolution . 2001,第3期

机译：线粒体突变可能会降低种群活力
2. 2,2′,4,4′-Tetrabromodiphenyl ether injures cell viability and mitochondrial function of mouse spermatocytes by decreasing mitochondrial proteins Atp5b and Uqcrc1 [J] . Shaoping Huang, Jing Wang, Yiqiang Cui Environmental toxicology and pharmacology . 2016,第SEPa期

机译：2,2'，4,4'-四溴二苯醚通过减少线粒体蛋白Atp5b和Uqcrc1损伤小鼠精母细胞的细胞活力和线粒体功能
3. Oxidative Insults and Mitochondrial DNA Mutation Promote Enhanced Autophagy and Mitophagy Compromising Cell Viability in Pluripotent Cell Model of Mitochondrial Disease [J] . Dar-Shong Lin, Yu-Wen Huang, Che-Sheng Ho, Cells . 2019,第1期

机译：氧化损伤和线粒体DNA突变促进线粒体疾病多能细胞模型中增强的自噬和线粒体损害细胞活力。
4. Mitochondrial DNA Mutations and Diseases [C] . Lee-Jun C. Wong Annual Meeting of the American College of Veterinary Pathologists . 2014

机译：线粒体DNA突变和疾病
5. The role of mitochondrial DNA mutations in sarcopenia: Implications for the mitochondrial "vicious cycle" theory and apoptosis. [D] . Hiona, Asimina. 2007

机译：线粒体DNA突变在少肌症中的作用：对线粒体“恶性循环”理论和细胞凋亡的影响。
6. Alexander disease causing mutations in the C-terminal domain of GFAP are deleterious both to assembly and network formation with the potential to both activate caspase 3 and decrease cell viability [O] . Yi-Song Chen, Suh-Ciuan Lim, Mei-Hsuan Chen, -1

机译：导致GFAP的C末端结构域突变的亚历山大疾病因组装和网络形成而有害其潜在的激活胱天蛋白酶3和降低细胞活力
7. Oxidative Insults and Mitochondrial DNA Mutation Promote Enhanced Autophagy and Mitophagy Compromising Cell Viability in Pluripotent Cell Model of Mitochondrial Disease [O] . Dar-Shong Lin, Yu-Wen Huang, Che-Sheng Ho, 2019

机译：氧化损伤和线粒体DNA突变促进线粒体疾病多能细胞模型中增强的自噬和水肿损害细胞活力

Mitochondrial mutations may decrease population viability

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