...
  • 主题
高级搜索  >
历史搜索 清空历史
首页> 外文期刊>Development >DEFECTIVE KERNEL 1 promotes and maintains plant epidermal differentiation
【24h】

DEFECTIVE KERNEL 1 promotes and maintains plant epidermal differentiation

机译：缺陷内核1促进和维持植物表皮分化

获取原文
获取原文并翻译 | 示例
           
页面导航
  • 摘要
  • 著录项
  • 相似文献
  • 相关主题

摘要

During plant epidermal development, many cell types are generated from protodermal cells, a process requiring complex co-ordination of cell division, growth, endoreduplication and the acquisition of differentiated cellular morphologies. Here we show that the Arabidopsis phytocalpain DEFECTIVE KERNEL 1 (DEK1) promotes the differentiated epidermal state. Plants with reduced DEK1 activity produce cotyledon epidermis with protodermal characteristics, despite showing normal growth and endoreduplication. Furthermore, in non-embryonic tissues (true leaves, sepals), DEK1 is required for epidermis differentiation maintenance. We show that the HD-ZIP IV family of epidermis-specific differentiation promoting transcription factors are key, albeit indirect, targets of DEK1 activity. We propose a model in which DEK1 influences HD-ZIP IV gene expression, and thus epidermis differentiation, by promoting cell adhesion and communication in the epidermis.
机译：在植物表皮发育期间，从原皮细胞产生许多细胞类型，该过程需要细胞分裂，生长，核内复制和分化细胞形态的获取的复杂协调。在这里，我们显示拟南芥植物钙蛋白酶缺陷内核1（DEK1）促进分化的表皮状态。 DEK1活性降低的植物产生具有原皮特征的子叶表皮，尽管显示出正常的生长和核内复制。此外，在非胚胎组织（真叶，萼片）中，DEK1是维持表皮分化所必需的。我们显示，表皮特异性分化的HD-ZIP IV家族促进转录因子是DEK1活性的关键（尽管是间接的）目标。我们提出了一种模型，其中DEK1通过促进表皮中的细胞粘附和通讯来影响HD-ZIP IV基因表达，从而影响表皮分化。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱：kefu@zhangqiaokeyan.com

京公网安备：11010802029741号 ICP备案号：京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有

  • 客服微信

  • 服务号