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首页> 外文期刊>Journal of athletic training >Factor v Leiden thrombophilia in a female collegiate soccer athlete: A case report
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Factor v Leiden thrombophilia in a female collegiate soccer athlete: A case report

机译：一名女大学生足球运动员的Factor v Leiden血栓形成症：一例报告

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Objective: To raise awareness among health care providers caring for an active population to an uncommon genetic mutation that increases the risk for a potentially fatal venous thromboembolism. Background: A 19-year-old previously healthy female collegiate soccer athlete complained of coughing and progressively decreased exercise tolerance, which were attributed to a recent illness and lack of sleep. Later that evening, she complained of dyspnea and pleuritic pain and was referred to the emergency department. Bilateral pulmonary emboli were identified with computed tomography, and a hypercoagulable panel revealed that the patient was heterozygous for the factor V Leiden mutation. Differential Diagnosis: Pneumonia, pneumothorax, pericarditis, pleuritis, gastroesophageal reflux disease, pulmonary embolism. Treatment: Intravenous heparin therapy was initiated immediately in the emergency department. This was followed by inpatient anticoagulant therapy for 5 days and outpatient anticoagulant therapy for an additional 12 months. During this time, the patient was unable to participate in soccer drills or return to competition and was limited to conditioning activities due to the risk of increased bleeding time. Uniqueness: Documented cases of pulmonary embolism in a young athletic population are rare and are usually associated with genetic risk factors. Factor V Leiden is a relatively uncommon genetic mutation that dramatically increases the risk for venous thromboembolism. Although the fatality rate in this population is low, fatality is preventable if the condition is recognized early and managed properly. Conclusions: Athletes should be encouraged to communicate with their athletic trainers regarding any changes in health status or medication usage. When an athlete presents with nonspecific symptoms such as dyspnea and chest pain, athletic trainers should consider the possibility of pulmonary embolism. A high degree of suspicion results in early diagnosis and treatment and may prevent a fatal event.
机译：目的：提高医疗保健提供者的意识，他们照顾活跃的人群发生罕见的基因突变，从而增加了潜在致命静脉血栓栓塞的风险。背景：一名19岁以前健康的女大学生足球运动员抱怨咳嗽并逐渐降低了运动耐力，这归因于最近患病和睡眠不足。那天晚上晚些时候，她抱怨呼吸困难和胸膜炎疼痛，并被转诊到急诊科。通过计算机断层扫描确定双侧肺栓塞，高凝组显示该患者杂合了V因子Leiden突变。鉴别诊断：肺炎，气胸，心包炎，胸膜炎，胃食管反流病，肺栓塞。治疗：急诊科立即开始静脉肝素治疗。随后是住院抗凝治疗5天，门诊抗凝治疗另外12个月。在这段时间内，患者由于无法增加出血时间而无法参加足球训练或重新参加比赛，并且只能进行调理活动。独特性：在年轻的运动人群中，肺栓塞的记录病例很少，通常与遗传危险因素有关。因子V莱顿是相对罕见的基因突变，可大大增加静脉血栓栓塞的风险。尽管该人群的死亡率很低，但如果及早发现并妥善处理，可以避免死亡。结论：应鼓励运动员就健康状况或药物使用的任何变化与体育教练进行沟通。当运动员出现呼吸道不适和胸痛等非特异性症状时，运动教练应考虑发生肺栓塞的可能性。高度怀疑会导致早期诊断和治疗，并可能预防致命事件。

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