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首页> 外文期刊>Biochimica et Biophysica Acta. Molecular and cell biology of Lipids >Dietary coconut oil increases conjugated linoleic acid-induced body fat loss in mice independent of essential fatty acid deficiency
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Dietary coconut oil increases conjugated linoleic acid-induced body fat loss in mice independent of essential fatty acid deficiency

机译：日粮椰子油增加了共轭亚油酸诱导的小鼠体内脂肪的损失，而与必需脂肪酸缺乏无关

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Conjugated linoleic acid (CLA) induces a body fat loss that is enhanced in mice fed coconut oil (CO), which lacks essential fatty acids (EFA). Our objective was to determine if CO enhancement of CLA-induced body fat loss is due to the lack of FFA. The CLA-EFA interaction was tested by feeding CO and fat free (FF) diets for varying times with and without replenishment of individual EFA, Mice fled CO during only the 2-week CLA-feeding period did not differ from control mice in their adipose EFA content but still tended ( P=0.06) to be leaner than mice fed soy oil (SO). Mice raised on CO or FF diets and fed CLA were leaner than the SO+CLA-fed mice (P < 0.01), Mice raised on CO and then replenished with linoleic, linolenic, or arachidonic acid were leaner when fed CLA than mice raised on SO (P < 0.001), Body fat of CO4 CLA-fed mice was not affected by EFA addition. In summary, CO-fed mice not lacking in tissue EFA responded more to CLA than SO-fed mice. Also, EFA addition to CO diets did not alter the enhanced response to CLA. Therefore, the increased response to CLA in mice raised on CO or FF diets appears to be independent of a dietary EFA deficiency. (c) 2005 Elsevier B.V. All rights reserved.
机译：共轭亚油酸（CLA）引起的人体脂肪损失在喂食缺少必需脂肪酸（EFA）的椰子油（CO）的小鼠中得到增强。我们的目标是确定CLA诱导的体内脂肪损失的CO增强是否归因于缺乏FFA。 CLA-EFA交互作用通过在有和没有补充个别EFA的情况下喂养不同时间的CO和无脂肪（FF）饮食进行测试，小鼠仅在2周CLA喂养期间逃逸了CO，与对照组小鼠的脂肪没有区别EFA含量，但仍趋于（P = 0.06）较饲喂豆油（SO）的小鼠更瘦。用CO或FF日粮喂养和饲喂CLA的小鼠比用SO + CLA喂养的小鼠瘦（P <0.01），用CO饲喂，然后补充亚油酸，亚麻酸或花生四烯酸的小鼠饲喂CLA的小鼠比饲喂CLA的小鼠瘦。 SO（P <0.001），CO4 CLA喂养的小鼠的体脂不受EFA添加的影响。总之，没有组织EFA的CO喂养的小鼠对CLA的反应比SO喂养的小鼠要多。另外，在CO饮食中添加EFA不会改变对CLA的增强反应。因此，用CO或FF饮食饲养的小鼠对CLA的反应增强似乎与饮食中EFA缺乏无关。 （c）2005 Elsevier B.V.保留所有权利。

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