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首页> 外文期刊>Journal of clinical anesthesia >Dexmedetomidine controls twitch-convulsive syndrome in the course of uremic encephalopathy
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Dexmedetomidine controls twitch-convulsive syndrome in the course of uremic encephalopathy

机译:右美托咪定在尿毒症脑病过程中控制抽搐-抽搐综合征

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摘要

An 85 year old man with a history of chronic renal insufficiency was admitted to the cardiothoracic intensive care unit after aortic valve replacement. His postoperative course was marked by acute oliguric renal failure for high blood urea nitrogen (BUN) and acute hyperactive delirium. At this time he also developed tremors with muscle twitching; he received no other form of sedatives. A neurology consult made the diagnosis of twitch-convulsive syndrome associated with uremic encephalopathy. While the patient was receiving the dexmedetomidine infusion, the signs of the twitch-convulsive syndrome, particularly the twitching and tremors, disappeared. Within 30 minutes of the end of the dexmedetomidine infusion, symptoms of the twitch-convulsive syndrome returned, manifesting as acute tremulousness. After several dialysis treatments, his BUN decreased and the dexmedetomidine was weaned, without return of the symptoms of twitch-convulsive syndrome.
机译:一位有慢性肾功能不全病史的85岁男子在主动脉瓣置换后被纳入心胸重症监护病房。他的术后病程以高尿素氮(BUN)和急性多动性ir妄的急性少尿性肾衰竭为特征。这时他还因肌肉抽搐而发抖。他没有收到其他任何形式的镇静剂。一位神经病学专家对尿毒症脑病相关的抽搐-抽搐综合征进行了诊断。在患者接受右美托咪定输注的同时,抽搐-抽搐综合征的体征,特别是抽搐和震颤消失了。在右美托咪定输注结束后的30分钟内,抽搐综合征的症状再次出现,表现为急性颤抖。经过数次透析治疗后,他的BUN减少,右美托咪定断奶,而抽搐-抽搐综合征的症状没有恢复。

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