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首页> 外文期刊>Journal of Neurology, Neurosurgery and Psychiatry >Deterioration of pre-existing hemiparesis brought about by subsequent ipsilateral lacunar infarction.
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Deterioration of pre-existing hemiparesis brought about by subsequent ipsilateral lacunar infarction.

机译：继发的同侧腔隙性脑梗塞导致先前存在的偏瘫的恶化。

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摘要

Mechanisms of post-stroke recovery are still poorly understood. Recent evidence suggests that cortical reorganisation in the unaffected hemisphere plays an important role. A 59 year old man developed a small lacunar infarct in the left corona radiata, which then caused marked deterioration in a pre-existing left hemiparesis that had resulted from an earlier right putaminal haemorrhage. Functional magnetic resonance imaging showed that the paretic left hand grip activated the ipsilateral left motor areas, but not the right hemispheric motor areas. This suggests that partial recovery of the left hemiparesis had been brought about by cortical reorganisation of the left hemisphere and intensification of the uncrossed corticospinal tract. The subsequent small infarct may have damaged the uncrossed tract, thereby causing the pre-existing hemiparesis to deteriorate even further.
机译：卒中后恢复的机制仍知之甚少。最近的证据表明，未受影响的半球的皮质重组起着重要的作用。一名59岁的男子在左侧的日冕辐射中出现了一个小腔隙性梗塞，继而导致由先前的右侧肠壁出血引起的先前存在的左偏瘫明显恶化。功能性磁共振成像显示，左手的局部抓握激活了同侧的左运动区，但未激活右半球运动区。这表明左半身轻瘫的部分恢复是由左半球的皮质重组和未交叉的皮质脊髓束的强化引起的。随后的小梗塞可能已经损坏了未交叉的管道，从而导致先前存在的偏瘫进一步恶化。

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