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首页> 外文期刊>Current opinion in gastroenterology >Implications of diet on nonalcoholic fatty liver disease.
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Implications of diet on nonalcoholic fatty liver disease.

机译：饮食对非酒精性脂肪肝的影响。

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PURPOSE OF REVIEW: This review examines the effects of diet on nonalcoholic fatty liver disease (NAFLD). This includes the effects of calories, both in excess and restricted, as well as macronutrients. RECENT FINDINGS: Recent findings suggest that short-term hypercaloric feeding leads to increased intrahepatic triglyceride (IHTG), whereas short-term hypocaloric feeding leads to decreased IHTG, despite little change in total body weight, suggesting that ongoing excess caloric delivery directly contributes to the development of NAFLD. Weight loss with either low-fat or low-carbohydrate diets can improve IHTG; however, specific macronutrients, such as fructose, trans-fatty acids, and saturated fat, may contribute to increased IHTG independent of total calorie intake. n-3 polyunsaturated fatty acids and monounsaturated fatty acids may play a protective role in NAFLD. The mechanisms behind these effects are not fully understood. SUMMARY: Diet plays a role in the pathophysiology of NAFLD. It is reasonable to advise patients with NAFLD to reduce calorie intake with either low-fat or low-carbohydrate diets as well as limit intakes of fructose, trans-fatty acids, and saturated fat.
机译：审查目的：这项审查检查饮食对非酒精性脂肪肝疾病（NAFLD）的影响。这包括热量的过量和限制以及大量营养素的影响。最近的发现：最近的研究结果表明，尽管总体重变化不大，但短期高热量喂养会导致肝内甘油三酸酯（IHTG）升高，而短期低热量喂养会导致IHTG降低，这表明持续的热量输送直接导致了肝脏热量的增加。 NAFLD的发展。低脂饮食或低碳水化合物饮食可以减轻IHTG;但是，特定的大量营养素，例如果糖，反式脂肪酸和饱和脂肪，可能会导致IHTG增加，而与总卡路里摄入量无关。 n-3多不饱和脂肪酸和单不饱和脂肪酸可能在NAFLD中起保护作用。这些作用背后的机制尚未完全了解。摘要：饮食在NAFLD的病理生理中发挥作用。合理建议NAFLD患者减少低脂或低碳水化合物饮食的卡路里摄入，并限制果糖，反式脂肪酸和饱和脂肪的摄入。

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