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首页> 外文期刊>European journal of clinical investigation >Ventricular mechanics after repair of subarterial and perimembranous VSD VSD s
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Ventricular mechanics after repair of subarterial and perimembranous VSD VSD s

机译：蛛网膜术后的心室力学，VSD vsd vsd

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Abstract Background Emerging data suggest impaired biventricular function in adults late after repair of ventricular septal defect ( VSD ). We assessed and compared right ( RV ) and left ventricular ( LV ) mechanics in adolescents and adults after surgical closure of doubly committed subarterial and perimembranous VSD s. Methods A total of 75 subjects were studied: 29 patients after subarterial VSD repair (group I), 17 patients after perimembranous VSD repair (group II ) and 29 age‐matched controls (group III ). RV and LV mechanics were assessed using tissue Doppler and speckle‐tracking echocardiography, while RV outflow systolic function was quantified by systolic excursion and fractional shortening ( FS ). Results Compared with group III , groups I and II had significantly reduced tricuspid annular systolic and diastolic velocities, isovolumic myocardial acceleration, RV global longitudinal systolic and diastolic deformation parameters and RV outflow systolic excursion (all P .05). Group I, but not II , had reduced RV outflow FS ( P = .008) and the lowest global LV longitudinal systolic strain ( P = .008) and systolic strain rate ( P = .014). In group I, postoperative aortic regurgitation was associated with lower LV longitudinal systolic strain ( P = .009) and early diastolic strain rate ( P = .002), while right bundle branch block was associated with lower RV systolic strain rate ( P = .048). As a group, RV outflow excursion ( P .001) and FS ( P = .001) were correlated with LV global systolic strain. Conclusion Adolescents and adults late after repair of subarterial and perimembranous VSD s show impairment of RV systolic and diastolic myocardial deformation. The RV outflow function and LV systolic deformation appear to be worse after repair of subarterial defects.
机译：摘要背景新兴数据在维修心室隔膜缺损（VSD）后晚些时候在成年人中表达了患者的双心功能受损。我们在双层闭合的子线圈和周膜VSD S的手术闭合后评估和比较了青少年和成人的右心室（LV）力学。方法采用共75名受试者进行了75名受试者：29例患者后序流VSD修复（Ⅰ组），17例患者在宫外血管VSD修复（II组）和29岁匹配对照（第三组）。使用组织多普勒和斑点跟踪超声心动图评估RV和LV力学，而RV流出收缩功能由收缩偏移和分数缩短（FS）量化。结果与III族相比，I和II组显着降低了三尖瓣环状和舒张速度，内含性心肌加速度，RV全局纵向收缩性和舒张变形参数和RV流出收缩促进（所有P <.05）。 I族，但不是II，减少了RV流出FS（P = .008）和最低全球LV纵向收缩菌株（P = .008）和收缩菌株率（P = .014）。在I族中，术后主动脉反流与较低的LV纵向收缩菌株（P = .009）和早期舒张菌株（P = .002）有关，而右束分支块与较低的RV收缩率应变率相关（P =。 048）。作为组，RV流出偏移（P＆ .001）和Fs（p = .001）与LV全局收缩菌株相关。结论双层和成人后期修复下颌和概要的VSD S表明RV收缩和舒张心肌变形的损害。 RV流出功能和LV收缩变形似乎在修复子宫缺陷后更糟糕。

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