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机译:通过不同的亚细胞微粒瘤,RA和RAP信号双向突触塑性度
Department of Pharmacology University of Virginia School of Medicine;
Department of Pharmacology University of Virginia School of Medicine;
Department of Pharmacology University of Virginia School of Medicine;
Department of Microbiology University of Virginia School of Medicine;
Department of Pharmacology University of Virginia School of Medicine;
Department of Neuroscience Johns Hopkins University School of Medicine;
Department of Pharmacology University of Iowa;
School of Pharmaceutical Sciences Wenzhou Medical University;
Instituto de Biomedicina y Biotecnología de Cantabriaand CIBERONC Consejo Superior de;
Department of Pharmacology University of Iowa;
School of Pharmaceutical Sciences Wenzhou Medical University;
Department of Neuroscience Johns Hopkins University School of Medicine;
Department of Pharmacology University of Virginia School of Medicine;
AMPA-R trafficking; AMPA-R phorphorylation; GluA1; GluA2; GluA4; GluA2L; organelle fractionation; nanocluster; subcellular signaling;
机译:通过不同的亚细胞微粒瘤,RA和RAP信号双向突触塑性度
机译:Rap1将cAMP信号转导至一个独特的p42 / 44MAPK池,以调节兴奋性,突触可塑性,学习和记忆。
机译:大鼠内嗅皮层表层和深层中双向活动依赖性突触可塑性的不同机制。
机译:尖峰神经元中突触可塑性的紧凑的可重构混合信号实现
机译:Stargazin的C端PSD-95 / discs大/ zona occludens-1(PDZ)结合位点的磷酸化调节了其在双向突触可塑性中的运输。
机译:Ras和Rap通过不同的亚细胞微结构域信号双向突触可塑性
机译:Rap1耦合cAMP信号到不同的p42 / 44MAPK池,调节兴奋性,突触可塑性,学习和记忆