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机译:Langerhans细胞中的p38-信号传导促进了产生IL-17的产生T细胞和牛皮癣皮肤炎症的发育
Hongqiao International Institute of Medicine Shanghai Tongren Hospital/Facultyof Basic Medicine Shanghai Institute of Immunology Key Laboratory of CellDifferentiation and Apoptosis of Chinese Ministry of Education Shanghai JiaoTong University School o;
Hongqiao International Institute of Medicine Shanghai Tongren Hospital/Facultyof Basic Medicine Shanghai Institute of Immunology Key Laboratory of CellDifferentiation and Apoptosis of Chinese Ministry of Education Shanghai JiaoTong University School o;
Hongqiao International Institute of Medicine Shanghai Tongren Hospital/Facultyof Basic Medicine Shanghai Institute of Immunology Key Laboratory of CellDifferentiation and Apoptosis of Chinese Ministry of Education Shanghai JiaoTong University School o;
Hongqiao International Institute of Medicine Shanghai Tongren Hospital/Facultyof Basic Medicine Shanghai Institute of Immunology Key Laboratory of CellDifferentiation and Apoptosis of Chinese Ministry of Education Shanghai JiaoTong University School o;
Hongqiao International Institute of Medicine Shanghai Tongren Hospital/Facultyof Basic Medicine Shanghai Institute of Immunology Key Laboratory of CellDifferentiation and Apoptosis of Chinese Ministry of Education Shanghai JiaoTong University School o;
Xin Hua Hospital affiliated to Shanghai Jiao Tong University School of Medicine Shanghai 200092 China;
Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics Guangdong Medical University Dongguan 523808 China;
Shanghai Skin Disease Hospital Tongji University Shanghai 200443 China;
Department of Cardiovascular Medicine Graduate School of Medicine Osaka University Osaka 565-0871 Japan;
Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics Guangdong Medical University Dongguan 523808 China;
机译:Langerhans细胞中的p38-信号传导促进了产生IL-17的产生T细胞和牛皮癣皮肤炎症的发育
机译:IL-17C / IL-17受体E信号在CD4(+)T细胞中促进T(H)17细胞驱动的肾小球炎症
机译:表皮朗格汉斯细胞和真皮树突状细胞产生独特的和互补的细胞因子,维持活动性牛皮癣的皮肤炎症
机译:RABGEF1,RAS信号传导的负调节器,肥大细胞活化和皮肤炎症
机译:揭示朗格汉斯细胞的奥秘:从朗格汉斯细胞组织细胞增生到朗格汉斯细胞辐射抗性。
机译:皮肤γδT细胞不能自由地从皮肤中循环出来并产生IL-17来促进初级接触性超敏反应期间的中性粒细胞浸润
机译:Langerhans细胞中的P38α信号传导促进了产生IL-17的产生T细胞和牛皮癣皮肤炎症的发育