...
  • 主题
高级搜索  >
历史搜索 清空历史
首页> 外文期刊>Journal of genetics >encodes protein(s) and the altered RuBisCO could be the proteomics leader of hybrid necrosis in wheat (Triticum aestivum L.)
【24h】

encodes protein(s) and the altered RuBisCO could be the proteomics leader of hybrid necrosis in wheat (Triticum aestivum L.)

机译：编码蛋白质和改变的Rubisco可以是小麦杂交坏死的蛋白质组学领导者（Triticum aestivum L.）

获取原文
获取原文并翻译 | 示例
           
页面导航
  • 摘要
  • 著录项
  • 相似文献
  • 相关主题

摘要

Wheat hybrid necrosis is caused by the interaction of two dominant complementary genes, Ne1 and Ne2, located on chromosome arms 5BL and 2BS, respectively. The sequences of Ne1 or Ne2 have not yet been identified. It is also not known whether Ne1 and Ne2 are structural or regulatory genes. Understanding the proteomic pathways may provide a knowledge base for protecting or maximizing the photosynthesis capacity of wheat. Using DIGE and MALDI-TOF-TOF MS, the flag leaf protein patterns of the two unique near-isogenic line siblings (NILs), the necrotic ShunMai 12Ah (Ne1 Ne1 Ne2 Ne2) and the normal ShunMai 12Af (Ne1 Ne1 ne2 ne2) were compared. Due to the presence or absence of Ne2, (i) three protein spots were expressed or disappeared, (ii) seven RuBisCO-related proteins were altered significantly, and (iii) 21 photosynthesis/glucose related proteins were changed significantly. Three hypotheses were deduced, (i) Ne1 may also encode protein(s), (ii) genetic maladjustment of RuBisCO could lead to early leaf death, and (iii) interactions between nuclear genes and chloroplast genes could determine photosynthetic traits. Our hypothetical model presents the RuBisCO pathway of hybrid necrosis in wheat and explains how Ne1 and Ne2 interact at molecular level.
机译：小麦混合坏死是由位于染色体臂5BL和2BS上的两个显性互补基因，NE1和NE2的相互作用引起的。尚未识别NE1或NE2的序列。还不知道NE1和NE2是否是结构或调节基因。理解蛋白质组学途径可以提供用于保护或最大化小麦光合作用容量的知识库。使用Dige和Maldi-Tof-TOF MS，标志叶片蛋白质图案的两个独特的近代线兄弟（NILS），坏死的顺声12AH（NE1 NE1 NE2 NE2）和正常顺声12AF（NE1 NE1 NE2 NE2）是比较的。由于NE2的存在或不存在，（i）表达或消失了三种蛋白质斑点，（ii）七种相关的蛋白质显着改变，（iii）21光合作用/葡萄糖相关蛋白质显着变化。推导出三个假设，（I）NE1也可以编码蛋白质，（ii）鲁西哥的遗传调节可能导致早叶死亡，（III）核基因与叶绿体基因之间的相互作用可以确定光合状体性状。我们的假设模型在小麦中展示了杂交坏死的Rubisco途径，并解释了NE1和NE2如何在分子水平上相互作用。

著录项

  • 来源
    《Journal of genetics》 |2017年第2期|共11页
  • 作者

    Pan Si Rui; Pan Xing Lai; Pan Qianying; Shi Yin Hong; Zhang Li; Fan Yun; Xue Yan Rui;

  • 作者单位

    Beijing Univ Chem Technol Life Sci &

    Technol A1201 Beijing 102200 Peoples R China;

    Shanxi Agr Sci Acad Cotton Res Inst Dept Food Crop Sci Yuncheng 044000 Shanxi Peoples R China;

    Anhui Univ Technol &

    Sci Foreign Language Coll Wuhu 241000 Anhui Peoples R China;

    Shanxi Agr Sci Acad Cotton Res Inst Dept Food Crop Sci Yuncheng 044000 Shanxi Peoples R China;

    Shanxi Agr Sci Acad Cotton Res Inst Dept Food Crop Sci Yuncheng 044000 Shanxi Peoples R China;

    Shanxi Agr Sci Acad Cotton Res Inst Dept Food Crop Sci Yuncheng 044000 Shanxi Peoples R China;

    Shanxi Agr Sci Acad Cotton Res Inst Dept Food Crop Sci Yuncheng 044000 Shanxi Peoples R China;

  • 收录信息
  • 原文格式 PDF
  • 正文语种 eng
  • 中图分类 遗传学;
  • 关键词

    autoimmune; chlorenchyma; hypersensitive response; posttranslational behaviour; rbcS; rbcL;

    机译：自身免疫;咽部;过敏反应;后期行为;RBCS;RBCL;

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱：kefu@zhangqiaokeyan.com

京公网安备：11010802029741号 ICP备案号：京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有

  • 客服微信

  • 服务号