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首页> 外文期刊>Allergy >Bacteriuria increases the risk of edematous attacks in hereditary angioedema with C1-inhibitor deficiency
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Bacteriuria increases the risk of edematous attacks in hereditary angioedema with C1-inhibitor deficiency

机译：细菌性尿毒症增加患有C1抑制剂缺乏症的遗传性血管性水肿引起水肿发作的风险

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Urinary tract infections are considered among the most common infectious disorders in humans. Various infections may have a role in inducing HAE attacks. Our study intended to evaluate bacteriuria in the urinalysis of patients with C1-INH-HAE. Urine specimens contributed by 139 patients with C1-INH-HAE at the annual control visits were studied retrospectively for microorganisms. We analyzed the presence of bacteriuria in relation to the clinical symptoms. Taking into account three randomly selected urine specimens, we found that the cumulative number of edematous attacks was higher in patients with bacteriuria than in those without (P = 0.019, P = 0.022, P = 0.014). Considering the same patients, attack number was significantly higher (14.51 vs 8.63) in patients with bacteriuria than in those without (P < 0.0001). In patients with bacteriuria, we found a higher incidence of edema formation during the year before evaluation, which may suggest the triggering role of bacteriuria in the occurrence of edematous episodes.
机译：尿路感染被认为是人类最常见的传染性疾病。各种感染可能会导致HAE攻击。我们的研究旨在评估C1-INH-HAE患者尿液分析中的细菌尿症。回顾性研究了139例C1-INH-HAE患者在年度对照访视中贡献的尿液标本中的微生物。我们分析了与临床症状有关的细菌尿的存在。考虑到三个随机选择的尿液样本，我们发现细菌性尿毒症患者的水肿发作累积次数高于无细菌尿症的患者（P = 0.019，P = 0.022，P = 0.014）。考虑到同一患者，细菌尿患者的发作次数显着高于无细菌患者（14.51比8.63）（P <0.0001）。在细菌尿患者中，我们发现在评估前一年中水肿形成的发生率较高，这可能表明细菌尿在水肿发作中的触发作用。

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