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首页> 外文期刊>Annals of hematology >Lupus anticoagulant hypoprothrombinemia syndrome in Bence-Jones protein κ-type multiple myeloma patient with phosphatidylserine-dependent antiprothrombin antibody.
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Lupus anticoagulant hypoprothrombinemia syndrome in Bence-Jones protein κ-type multiple myeloma patient with phosphatidylserine-dependent antiprothrombin antibody.

机译：具有磷脂酰丝氨酸依赖性抗凝血酶原抗体的Bence-Jones蛋白κ型多发性骨髓瘤患者的狼疮抗凝性凝血酶原低血症综合征。

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摘要

Approximately 2 % of multiple myeloma (MM) patients present with hemorrhage at diagnosis. However, hemorrhage due to abnormalities in the coagulation system is a rare complication [1,2]. Although lupus anticoagulant (LA), which is infrequently reported to be in association with MM, is commonly a risk factor for arterial or venous thrombosis, bleeding tendencies in patients with LA are strongly related to a low prothrombin activity [3-6]. Acquired hypopro-thrombinemia with LA, also called LA hypoprothrombine-mia syndrome (LAHPS), is a rare disease which appears mostly in young females with systemic lupus erythematosus or in healthy children after viral infection and is usually associated with the presence of antiprothrombin antibodies [7].
机译：多发性骨髓瘤（MM）患者中约有2％在诊断时出现出血。然而，由于凝血系统异常而引起的出血是一种罕见的并发症[1,2]。尽管很少报道与MM相关的狼疮抗凝剂（LA）通常是动脉或静脉血栓形成的危险因素，但LA患者的出血倾向与凝血酶原活性低密切相关[3-6]。获得性LA患者的凝血酶原低血症，也称为LA凝血酶原低下综合征（LAHPS），是一种罕见疾病，多见于系统性红斑狼疮的年轻女性或病毒感染后的健康儿童中，通常与抗凝血酶原抗体的存在有关[ 7]。

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