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首页> 外文期刊>Acta Physiologiae Plantarum >Effect of salicylic acid potentiates cadmium-induced oxidative damage in Oryza sativa L. leaves
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Effect of salicylic acid potentiates cadmium-induced oxidative damage in Oryza sativa L. leaves

机译：水杨酸对镉胁迫下水稻叶片氧化损伤的增强作用

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In the present investigation, we studied the possible potentiating effect of salicylic acid (SA) under Cd toxicity in Oryza sativa L. leaves. Cd treatments for 24 h reduced the shoot length, dry biomass and total chlorophyll content followed by high Cd accumulation in shoots. About 16 h presoaking with SA resulted in partial protection against Cd, as observed by minor changes in length, bio-mass and total chlorophyll. SA priming resulted in low Cd accumulation. Enhanced thiobarbituric acid reactive substances (TEARS), hydrogen peroxide (H_2O_2) and super-oxide anion (O_2~-) content were seen when Cd was applied alone, while under SA priming the extent of TEARS, H_2O_2 and O_2~- were significantly low, suggesting SA-regulated protection against oxidative stress. The antioxidant enzymes like Catalase (CAT), guaiacol peroxidase (GPx), glutathione reductase (GR) and superoxide dismutase (SOD) showed varied activities under Cd alone. CAT activity increased after Cd treatment, followed by a decline in GPX and GR activity. SOD also declined at the highest concentrations with an initial increase. Under SA-priming conditions, the efficiency of the antioxidant enzymes was significantly elevated. GPx and SOD activity showed significant increase in activity.The ascorbate activity increased after Cd treatment, followed by a decline in glutathione under SA-free condition. SA priming showed gradual increase in these non-enzymic antioxidants. Our results indicate that Cd-induced oxidative stress can be regulated by SA.
机译：在本研究中，我们研究了水杨酸（SA）在水稻叶片中Cd毒性下可能的增强作用。镉处理24小时减少了芽的长度，干生物量和总叶绿素含量，随后使芽中的镉积累高。通过长度，生物量和总叶绿素的微小变化观察到，用SA预浸泡约16 h可以部分保护Cd。 SA引发导致Cd积累低。单独施用镉时，硫代巴比妥酸反应性物质（TEARS），过氧化氢（H_2O_2）和超氧阴离子（O_2〜-）含量增加，而在SA引发下，TEARS，H_2O_2和O_2〜-的含量显着降低，表明SA调节了针对氧化应激的保护作用。像过氧化氢酶（CAT），愈创木酚过氧化物酶（GPx），谷胱甘肽还原酶（GR）和超氧化物歧化酶（SOD）等抗氧化酶在单独的Cd下表现出不同的活性。镉处理后CAT活性增加，随后GPX和GR活性下降。 SOD在最高浓度时也有所下降，并开始上升。在SA引发条件下，抗氧化酶的效率显着提高。 GPx和SOD活性显示出明显的活性增加.Cd处理后抗坏血酸活性增加，然后在无SA条件下谷胱甘肽下降。 SA引发显示这些非酶类抗氧化剂逐渐增加。我们的结果表明，镉诱导的氧化应激可以通过SA调节。

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