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首页> 外文期刊>Life sciences >Effect of the quassinoids glaucarubolone and simalikalactone D on growth of cells permanently infected with feline and human immunodeficiency viruses and on viral infections.
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Effect of the quassinoids glaucarubolone and simalikalactone D on growth of cells permanently infected with feline and human immunodeficiency viruses and on viral infections.

机译：quassinoids glaucarubolone和simalikalactone D对永久感染猫和人类免疫缺陷病毒的细胞生长以及病毒感染的影响。

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摘要

Growth of Crandall feline kidney cells permanently infected with feline immunodeficiency virus was inhibited by the anti-cancer quassinoid glaucarubolone whereas growth of uninfected cells was not inhibited. Similar results were obtained for human MOLT-4 cells infected with HIV-1. The results suggest that cell lines permanently infected with either the feline or the human lentivirus exhibit growth response characteristics to the quassinoids in common with other cell lines malignantly transformed. In addition the quassinoids may delay viral infection suggesting some commonality between the mechanism responsible for inhibition of the growth of the transformed phenotype and viral infection.
机译：永久性被猫免疫缺陷病毒感染的Crandall猫肾细胞的生长受到抗癌类拟古芥青草酮的抑制，而未感染细胞的生长未受到抑制。对于感染了HIV-1的人MOLT-4细胞，获得了相似的结果。结果表明，与恶性转化的其他细胞系一样，被猫或人慢病毒永久感染的细胞系表现出对类胡萝卜素的生长应答特性。此外，类胡萝卜素可能会延迟病毒感染，提示抑制转化表型生长的机制与病毒感染之间存在某些共性。

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