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机译:通过水解激活的产氧生物材料防止低氧诱导的β细胞和胰岛细胞死亡
Diabetes Research Institute and Departments of University of Miami, Miami, FL 33136,Department of Biomedical Engineering, College of Engineering, University of Miami, Coral Gables, FL 33134;
Diabetes Research Institute and Departments of University of Miami, Miami, FL 33136,Department of Biomedical Engineering, College of Engineering, University of Miami, Coral Gables, FL 33134;
Diabetes Research Institute and Departments of University of Miami, Miami, FL 33136,Department of Biomedical Engineering, College of Engineering, University of Miami, Coral Gables, FL 33134;
Diabetes Research Institute and Departments of University of Miami, Miami, FL 33136,Department of Biomedical Engineering, College of Engineering, University of Miami, Coral Gables, FL 33134,Surgery University of Miami, Miami, FL 33136,Medicine, Leonard M. Miller School of Medicine, University of Miami, Miami, FL 33136;
Diabetes Research Institute and Departments of University of Miami, Miami, FL 33136,Department of Biomedical Engineering, College of Engineering, University of Miami, Coral Gables, FL 33134,SurgeryUniversity of Miami, Miami, FL 33136;
tissue engineering; encapsulation; bioartificial pancreas; diabetes;
机译:PI3K信号的激活阻止了鼠耳蜗中氨基糖苷诱导的毛细胞死亡。PI3K信号的激活阻止了鼠耳蜗中氨基糖苷诱导的毛细胞死亡。
机译:Spirulina Maxima提取物通过对淀粉样蛋白β1-42(β(1-42))在PC12细胞中诱导神经毒性的BDNF活化来防止细胞死亡
机译:β细胞选择性K(ATP)通道激活保护β细胞和人类胰岛免受人类胰岛淀粉样多肽诱导的毒性作用。
机译:通过产生氧气的微珠预防缺氧诱导的β细胞死亡和功能障碍
机译:雌激素受体激活抑制胰岛的脂肪生成并减少脂质积累,并防止脂毒性β细胞衰竭
机译:通过水解激活的可产生氧气的生物材料防止低氧诱导的β细胞和胰岛细胞死亡
机译:通过水解活化的氧气产生生物材料预防β细胞和胰岛中缺氧诱导的细胞死亡