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首页> 外文期刊>DMW: Deutsche Medizinische Wochenschrift >67-year-old patient with speech disorder and dysphagia
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67-year-old patient with speech disorder and dysphagia

机译：67岁的语言障碍和吞咽困难患者

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A 67-year-old man who had been heart transplanted ten years before was admitted to our hospital because of diarrhea. During his stay he developed a severe lingual and facial angioedema. After excluding hereditary angioedema caused by a deficiency in functional C1 esterase inhibitor we focused on adverse effects of his drugs. The medication was composed of Aspirin, Enalapril, Diltiazem, Everolimus, Mycophenolate Mofetil, Bisoprolol, Pravastatin, Esomeprazol and Allopurinol. The angioedema disappeared with anti-allergic treatment. The administration of the suspected trigger enalapril was stopped. However weeks later the patient was admitted again with angioedema. Due to missing urticaria Aspirin was unlikely the causer. The only new drug the patient had been medicated with was Everolimus for 30 days. We assumed a link between the angioedema and Everolimus. Consequently we changed the immunosuppressive regime. After stopping Everolimus no angioedema occurred. Conclusion: Everolimus is a potential trigger of angioedema.
机译：一名十年前曾接受心脏移植手术的67岁男子因腹泻入院。在他逗留期间，他出现了严重的舌和面部血管性水肿。在排除由功能性C1酯酶抑制剂缺乏引起的遗传性血管性水肿后，我们集中研究了他的药物的不良反应。该药物由阿司匹林，依那普利，地尔硫卓，依维莫司，霉酚酸酯，比索洛尔，普伐他汀，埃索美拉唑和别嘌呤醇组成。经过抗过敏治疗，血管水肿消失了。停止给予可疑的触发依那普利。但是几周后，患者再次因血管水肿入院。由于缺少荨麻疹，阿司匹林不太可能是病因。患者服用的唯一新药是依维莫司30天。我们假设血管性水肿和依维莫司之间存在联系。因此，我们改变了免疫抑制方案。停止依维莫司后，未发生血管性水肿。结论：依维莫司可能是血管性水肿的潜在诱因。

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