• 主题
高级搜索  >
历史搜索 清空历史
首页> 外文期刊>Journal of stroke and cerebrovascular diseases: The official journal of National Stroke Association >Differential lesion patterns on T2-weighted magnetic resonance imaging and fluid-attenuated inversion recovery sequences in cryptogenic stroke patients with patent foramen ovale
【24h】

Differential lesion patterns on T2-weighted magnetic resonance imaging and fluid-attenuated inversion recovery sequences in cryptogenic stroke patients with patent foramen ovale

机译：卵圆孔未闭的隐源性卒中患者的T2加权磁共振成像和液体衰减反转恢复序列的差异病变模式

获取原文
获取原文并翻译 | 示例
           
页面导航
  • 摘要
  • 著录项
  • 相似文献
  • 相关主题

摘要

Background The present study aimed to determine lesion patterns and the stroke mechanisms in cryptogenic ischemic stroke patients with patent foramen ovale (PFO) on T2-weighted magnetic resonance imaging (T2WI) and fluid-attenuated inversion recovery (FLAIR) sequences combined. Methods In this retrospective study, 38 patients with cryptogenic stroke and an isolated PFO compared with 51 cryptogenic stroke patients without PFO were evaluated and their characteristics of lesion patterns on T2WI and FLAIR sequences combined were investigated. The number, distribution of small ischemic lesions, and the frequency of multiple small ischemic lesions were analyzed between the 2 groups. Results Thirty-two of 38 patients had a total of 341 small ischemic lesions in cryptogenic stroke patients with PFO versus 24 of 51 patients with 156 small ischemic lesions in patients without PFO, and, 8.97 ± 7.91 and 3.19 ± 4.82 ischemic lesions per person, respectively. Multiple small ischemic lesions occurred more frequently in cryptogenic stroke patients with PFO (25 of 38 patients, 66%) than in patients without PFO (16 of 51 patients, 31%; P =.001). Subcortical frontal and parietal small lesions were more frequent in cryptogenic stroke patients with PFO (28 of 38 patients, 74%) than in patients without PFO (18 of 51 patients, 35%; P <.0001). Conclusions Multiple small ischemic lesions and subcortical frontal and parietal small lesions were significantly associated with cryptogenic stroke patients with PFO, suggesting that paradoxical embolism may be the mechanism of PFO-associated cryptogenic stroke patients.
机译：背景技术本研究旨在确定合并T2加权磁共振成像（T2WI）和体液衰减倒置恢复（FLAIR）序列的卵圆孔未闭（PFO）的隐源性缺血性中风患者的病变模式和中风机制。方法在这项回顾性研究中，对38例隐源性卒中和孤立的PFO患者与51例无PFO的隐源性卒中患者进行了评估，并研究了其在T2WI和FLAIR序列上的病变模式特征。分析两组之间的小缺血性病变的数量，分布以及多发性小缺血性病变的频率。结果38例患者中有32例患有PFO的隐源性卒中患者共341例小缺血性病变，而无PFO的51例156个小缺血性病变的51例患者中有24例，每人分别为8.97±7.91和3.19±4.82缺血性病变，分别。与PFO无关的隐源性卒中患者中，多发性小缺血性病变发生率更高，而无PFO的患者（38例患者中的25例，占66％）比无PFO的患者（51例患者中的16例，占31％; P = .001）更常见。隐源性卒中合并PFO的患者（38例，28例，74％）比无PFO患者（51例中的18例，35％; P <.0001）更为常见。结论多发性小缺血性病变以及皮质下额叶和顶叶小病变与隐匿性卒中合并PFO有显着相关性，提示悖论性栓塞可能是PFO伴发隐匿性卒中的机制。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱：kefu@zhangqiaokeyan.com

京公网安备：11010802029741号 ICP备案号：京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有

  • 客服微信

  • 服务号