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首页> 外文期刊>Journal of Experimental Botany >Exposure to heavy metal stress triggers changes in plasmodesmatal permeability via deposition and breakdown of callose
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Exposure to heavy metal stress triggers changes in plasmodesmatal permeability via deposition and breakdown of callose

机译：暴露于重金属应力触发通过沉积和胼uding的沉积分离等离离子体渗透性的变化

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摘要

Both plants and animals must contend with changes in their environment. The ability to respond appropriately to these changes often underlies the ability of the individual to survive. In plants, an early response to environmental stress is an alteration in plasmodesmatal permeability with accompanying changes in cell to cell signaling. However, the ways in which plasmodesmata are modified, the molecular players involved in this regulation, and the biological significance of these responses are not well understood. Here, we examine the effects of nutrient scarcity and excess on plasmodesmata-mediated transport in the Arabidopsis thaliana root and identify two CALLOSE SYNTHASES and two beta-1,3-GLUCANASES as key regulators of these processes. Our results suggest that modification of plasmodesmata-mediated signaling underlies the ability of the plant to maintain root growth and properly partition nutrients when grown under conditions of excess nutrients.
机译：植物和动物都必须与其环境的变化相悖。 适当地对这些变化进行响应的能力往往使个人生存的能力成为基础。 在植物中，对环境压力的早期反应是具有伴随细胞对细胞信号传导的伴随变化的等离子体衍射性的改变。 然而，改变了诸如诸如此调节的分子参与的分子参与的方式，以及这些反应的生物学意义尚不清楚。 在这里，我们研究养分稀缺和过量对拟南芥中介导的转运的营养稀缺和过量的影响，并将两种胼ins-1,3-葡聚糖酶鉴定为这些过程的关键调节剂。 我们的研究结果表明，在多余的营养素的条件下，植物介导的信号传导的改性是植物在种植的条件下在生长时保持根系生长和适当分区营养的能力。

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