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首页> 外文期刊>Environmental Science and Pollution Research >Exogenously applied calcium alleviates cadmium toxicity in Matricaria chamomilla L. plants
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Exogenously applied calcium alleviates cadmium toxicity in Matricaria chamomilla L. plants

机译：外源施用的钙减轻了母菊植物中的镉毒性。

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Cadmium (Cd) toxicity in plants leads to serious disturbances of physiological processes, such as inhibition of chlorophyll synthesis, oxidative injury to the plant cells and water and nutrient uptake. Response of Matricaria chamomilla L, to calcium chloride (CaCl_2) enrichment in growth medium for reducing Cd toxicity were studied in this study. Hydroponically cultured seedlings were treated with 0,0.1,1, and 5 mM CaCl_2, under 0,120, and 180 hM CdCl2 conditions, respectively. The study included measurements pertaining to physiological attributes such as growth parameters, Cd concentration and translocation, oxidative stress, and accumulation of phenolics. Addition of CaCl_2 to growth media decreased the Cd concentration, activity of antioxi-dant enzymes, and reactive oxygen species accumulation in the plants treated with different CdCl_2, but increased the growth parameters. Malondialdehyde and total phenolics in shoots and roots were not much affected when plants were treated only with different CaCl_2 levels, but it showed a rapid increase when the plants were exposed to 120 and 180 CdCl2 levels. CaCl_2 amendment also ameliorated the CdCLrinduced stress by reducing oxidative injury. The beneficial effects of CaCl_2 in ameliorating CdCl_2 toxicity can be attributed to the Ca-induced reduction of Cd concentration, by reducing the cell-surface negativity and competing for Cd~+ ion influx, activity enhancement of antioxidant enzymes, and biomass accumulation.
机译：植物中的镉（Cd）毒性会严重破坏生理过程，例如抑制叶绿素合成，对植物细胞的氧化损伤以及水分和养分吸收。本研究研究了洋甘菊花L对生长培养基中氯化钙（CaCl_2）富集的响应，以降低Cd毒性。分别在0.120和180 hM CdCl2条件下，分别用0.1、0.1和5 mM CaCl_2处理水培苗。该研究包括与生理属性有关的测量，例如生长参数，Cd浓度和易位性，氧化应激和酚类物质的积累。在生长培养基中添加CaCl_2可以降低不同CdCl_2处理的植物中的Cd浓度，抗氧化酶活性和活性氧的积累，但增加了生长参数。当仅用不同的CaCl_2水平处理植物时，芽和根中的丙二醛和总酚含量受到的影响不大，但是当植物暴露于120和180 CdCl2水平时，其显示出快速增加。 CaCl_2修饰剂还通过减少氧化损伤减轻了CdCLr诱导的应激。 CaCl_2改善CdCl_2毒性的有益作用可归因于Ca诱导的Cd浓度降低，其方式是降低细胞表面负电荷并竞争Cd〜+离子流入，抗氧化剂酶活性增强和生物量积累。

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